• Daniel Águila-Gordo, José Manuel Flores-Barragán, Ferran Ferragut-Lloret, Jaime Portela-Gutierrez, Beatriz LaRosa-Salas, Lourdes Porras-Leal, and Carlos Villa GuzmánJoséJOncology Service of Hospital General de Ciudad Real, Spain..
• Cardiology Department, University Hospital of Ciudad Real, Obispo Torija s/n, 13005, Ciudad Real, Spain. Electronic address: danielaguilagordo@gmail.com.
• J Clin Neurosci. 2020 Oct 1; 80: 280-281.

AbstractThe etiological agent of coronavirus disease-19 (COVID-19), SARS-coronavirus-2 (SARS-CoV-2), emerged in Wuhan, China, and quickly spread worldwide leading the World Health Organization (WHO) to recognize it not only as a pandemic but also as an important thread to public health. Beyond respiratory symptoms, new neurological manifestations are being identified such as headache, ageusia, anosmia, encephalitis or acute cerebrovascular disease. Here we report the case of an acute transverse myelitis (TM) in a patient with SARS-CoV-2 infection detected by the nasopharyngeal swab technique but not in cerebrospinal fluid (CSF) analysis. Anti-herpes simplex virus (HSV) 1 and varicella-zoster IgM antibodies were not detected in serum samples and spinal and brain magnetic resonance imaging (MRI) showed no abnormal findings. This case remarks that COVID-19 nervous system damage could be caused by immune-mediated mechanisms.Copyright © 2020 Elsevier Ltd. All rights reserved.

39 keywords...

hide…