• F Gutiérrez Moreno, M L Rubio Montañés, J C Buñuel Alvarez, and M Guillén Galve.
• Medicina Familiar y Comunitaria, Zaragoza.
• Med Clin Barcelona. 1991 Oct 26; 97 (14): 531532531-2.

BackgroundThe nicotine contained in tobacco smoke produces an elevation of the plasmatic catecholamines which, in turn, may be the cause of the increase in basal glycemia by several mechanisms. The aim of this study was to investigate the relation between cigarette consumption and the increase in glucose in smokers and non smokers.MethodsBasal glycemia and glucosylate hemoglobin (HbA1c) were determined in 74 males, 38 smokers and 36 non smokers who were consulted and included in the prevention activities program of the center.ResultsSignificantly greater fasting glycemia was found in smokers than in nonsmokers. However, upon analyzing the values of glucosylate hemoglobin no significant differences were observed between the two groups.ConclusionsThese data suggest that nicotine contained in tobacco smoke produces a transitory hyperglycemia.

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