• W Winkelmüller.
• Klinik und Gemeinschaftspraxis für Neurochirurgie, Paracelsus-Klinik Osanbrück.
• Neurochirurgia (Stuttg). 1990 Mar 1;33(2):54-7.

AbstractThe trigeminal neuralgia has to be differentiated from the trigeminal neuropathy in respect of pain character and etiology. The neuralgia is characterized by paroxysmal pain evoked by trigger stimuli. The most frequent cause of this type of pain is a parapontine vascular compression of the trigeminal root without neurological deficits. The symptoms of neuropathy are some sensory loss associated with continuous pain resulting from peripheral damage of the trigeminal nerve. This distinction alleviates the indication for specific operative procedures and is more precise than the subdivision in typical and atypical neuralgia. The results of neurovascular decompression and thermorhizotomy can be much improved if neuropathic pain syndromes are excluded from operation. Out of 180 patients suffering from trigeminal neuralgia 94% were pain-free after neurovascular decompression and 96% of 144 patients following thermorhizotomy. For the treatment of continuous neuropathic pain augmentative electrostimulation of the Gasserian Ganglion via implanted electrodes is recommended.

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