• Esther Garazi, Suzanne Bridge, Anthony Caffarelli, Stephen Ruoss, and Pieter Van der Starre.
• From the Departments of *Anesthesiology, Perioperative and Pain Medicine, and ‖Pulmonary and Critical Care Medicine, Stanford University Medical Center, Stanford, California; †Department of Critical Care, Lakeridge Health, Oshawa, Ontario, Canada; ‡Department of Medicine, Queen's University, Ontario, Canada; and §Department of Cardiovascular Surgery, Hoag Memorial Hospital, Newport Beach, California.
• A A Case Rep. 2015 Jan 15;4(2):22-5.

AbstractSuccessful glycemic control reduces morbidity and mortality in cardiac surgery patients. Protocols that include insulin infusions are commonly followed to achieve target blood glucose levels. Insulin resistance has been reported and linked to low serum phosphate levels in animal models and studies in diabetic outpatients, but not in postoperative patients. The following case series is a retrospective observational review of 8 cardiac surgery patients who developed insulin resistance early after surgery; this resistance was reversed by correcting serum hypophosphatemia. We discuss the multiple underlying mechanisms causing hypophosphatemia.

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