• Jonathan Hulin, Pierre Aslanian, Georges Desjardins, Mustapha Belaïdi, and André Denault.
• From the *Department of Anesthesiology and Critical Care, Division of the Montreal Heart Institute, Université de Montréal, Montreal, Quebec, Canada; †Critical Care Division of the Centre Hospitalier de l'Université de Montréal, Montreal, Quebec; and ‡Department of Anesthesiology, Centre Hospitalier Universitaire de Nantes, Nantes, France.
• A A Case Rep. 2016 Mar 1; 6 (5): 114-20.

AbstractHepatic venous blood flow can be easily obtained using bedside ultrasound with either transthoracic or transesophageal echocardiography. Six critically ill patients with shock associated with absent or significantly reduced hepatic venous blood flow in the presence of normal or increased pulmonary venous flow are presented. In all these patients, the etiology of shock was secondary to increased resistance to venous return from either an intraabdominal process or through extrinsic or intrinsic occlusion of the proximal inferior vena cava or right atrium. These shock situations are secondary to increased resistance to venous return. Their treatment is highly specific and typically involves a surgical intervention.

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