Clinics in perinatology
-
Bronchopulmonary dysplasia, defined as ventilator or oxygen dependence accompanied by characteristic x-ray changes, occurs frequently in the tiny baby. The authors report their own experience in a large metropolitan hospital. A review of etiologic factors and therapeutic interventions for bronchopulmonary dysplasia in tiny infants is detailed.
-
Clinics in perinatology · Jun 1986
Resuscitation and respiratory management of infants weighing less than 1000 grams.
Care of the tiny neonate with regard to assisted ventilation and possible surfactant therapy is discussed in this review. Management in the delivery room and after is also included.
-
When mean arterial pressure exceeds 140 mmHg (equivalent to 180/120), there is a significant risk of maternal cerebral vascular damage. Therefore it is recommended that blood pressures greater than 170/110 should be treated with urgency, aiming to maintain the blood pressure at all times at less than 170/110 but not lower than 130/90. Parenteral hydralazine is effective and safe therapy. ⋯ There seems, therefore, to be no definite indication for treatment of mild hypertension in pregnancy; treatment of moderate hypertension may be reasonable but its value is unproved at present. Antihypertensive drugs are valuable in pregnancy to reduce the risks directly due to elevated blood pressure. These drugs are not expected to affect the evolution of preeclampsia nor to treat the other complications of this condition.
-
The maternal cardiovascular system undergoes extensive changes during pregnancy. These changes become especially important in women with underlying cardiac disease. In order to provide a basis for understanding cardiac disease in pregnancy, the authors provide a discussion of the normal cardiovascular physiology of pregnancy, followed by a review of the types of heart disease in women of childbearing age.
-
Discussed in this article are the three main anatomic types of persistent pulmonary hypertension of the newborn; (1) maladaptation of the pulmonary vascular bed; (2) excessive muscularization of the pulmonary vascular bed; and (3) underdevelopment of the pulmonary vascular bed.