Klinische Monatsblätter für Augenheilkunde
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Klin Monbl Augenheilkd · Nov 1989
[Oculo-oscillodynamography findings in glaucoma without hypertension].
Oculo-oscillodynamography after Ulrich was performed in 27 patients suffering from glaucoma without hypertension (so-called low-tension glaucoma). Patients who had severe systemic disease or were receiving systemic medication which might influence IOP, and patients with narrow angle and an IOP higher than 22 mm Hg were excluded. In 85% of the patients a severe decrease in systolic ciliary perfusion pressure was found, whereas the systolic retinal and diastolic ocular perfusion pressures were significantly lower in almost 30% of the cases. Glaucoma without hypertension appears to be caused by the vascular change at the disk.
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Klin Monbl Augenheilkd · Feb 1989
Randomized Controlled Trial Comparative Study Clinical Trial[Preoperative use of suction cup oculopression in comparison with Vörösmarthy oculopression].
In contrast to Vörösmarthy oculopression (VOP), suction-cup oculopression (SOP) is "pure" oculopression without compression of orbital tissue. Prior to 44 cataract operations with posterior chamber lens implantation, the authors performed SOP (negative pressure - 110 mm Hg) or VOP (level of oculopression 30 mm Hg) of the same duration (average 26 min). Before oculopression, a retrobulbar injection (RBI) was performed for local anesthesia. ⋯ After RBI + SOP, a post-injection of anesthetic was necessary in three cases; no additional injection was required after RBI + VOP. In conclusion, identical preoperative reductions in IOP can be achieved with SOP and VOP. Regarding the form of the iris-vitreous diaphragm and the frequency of post-injection, the differences between the two methods may be related to the lack of orbital compression during SOP.
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In the study reported here the authors investigated the influence on ocular perfusion pressures of suction-cup oculopression, a procedure which permits oculopression without orbital compression. Patients over 50 years of age were examined before cataract extraction. Ulrich's method of ocular oscillodynamography was used to determine ocular perfusion pressures. ⋯ However, ocular blood pressure remains constant after oculopression. Moreover, the increase in ocular perfusion pressure could not be explained by oculocardiac reflexes, because there was no change in systemic arterial blood pressure. It must be assumed, therefore, that the increase in ocular perfusion pressures is caused by a lowering of resistance, i.e., a drop in intraocular pressure.
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Klin Monbl Augenheilkd · Oct 1985
[Ulrich oculo-oscillodynamography: initial results in carotid stenoses].
Oculo-oscillodynamography (OODG) after Ulrich was performed in 11 patients suffering from a monolateral, hemodynamically relevant carotid stenosis identified by Doppler sonography. All of the pressure parameters which can be measured by OODG--systolic retinal, systolic ciliary, and diastolic ocular perfusion pressure--were low on the side concerned. In contrast to the already-established methods of measuring ocular perfusion--ophthalmodynamometry and ophthalmodynamography after Hager--the advantages of OODG are that it not only enables the systolic retinal pressure to be differentiated from the systolic ciliary or diastolic ocular perfusion pressure, but also permits the results of the investigation to be illustrated graphically, so that objective evaluation is possible.
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Ocular muscle diseases fall into two distinct groups: acute or subacute exophthalmic myositis and chronic oligosymptomatic myositis. The diagnosis can only be established with certainty by means of B-scan sonography and CT scanning; these examinations demonstrate the irregular thickening of one or several eye muscles. ⋯ The course is spontaneous, with continual recurrence, and may have sequelae in the form of a definitive exophthalmos or an acquired retraction syndrome. It responds remarkably well to corticotherapy and, if necessary, immunosuppressors; in general, these lead to rapid and - if treatment is started in time - total regression of the disease.