Journal of the American College of Cardiology
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J. Am. Coll. Cardiol. · Oct 1994
Evidence of reduced respiratory muscle endurance in patients with heart failure.
We sought to investigate whether reduced respiratory muscle endurance contributes to increased dyspnea and decreased exercise capacity in patients with chronic heart failure. ⋯ Respiratory muscle endurance as assessed by maximal sustainable ventilatory capacity is reduced in patients with heart failure.
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J. Am. Coll. Cardiol. · Sep 1994
T wave "humps" as a potential electrocardiographic marker of the long QT syndrome.
This study attempted to determine the prevalence and electrocardiographic (ECG) lead distribution of T wave "humps" (T2, after an initial T wave peak, T1) among families with long QT syndrome and control subjects. ⋯ These findings are consistent with the hypothesis that in families with long QT syndrome, T wave humps involving left precordial or (especially) limb leads, even among asymptomatic blood relatives with a borderline QTc interval, suggest the presence of the long QT syndrome trait.
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J. Am. Coll. Cardiol. · Sep 1994
Left ventricular wall motion abnormalities in patients with subarachnoid hemorrhage: neurogenic stunned myocardium.
The purpose of this study was to determine whether a relation exists between electrocardiographic (ECG) abnormalities and left ventricular wall motion in patients with subarachnoid hemorrhage. ⋯ These findings suggest that patients with subarachnoid hemorrhage and ST segment elevation may demonstrate transient corresponding regional wall motion abnormalities. The mechanism of neurogenic stunned myocardium was not clearly elucidated in the present study.
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J. Am. Coll. Cardiol. · Sep 1994
Beta-adrenergic blockade reduces myocardial injury during experimental cardiopulmonary resuscitation.
We attempted to determine the effects of beta-adrenergic blockade during cardiopulmonary resuscitation (CPR) on defibrillation rates and postresuscitation left ventricular function. ⋯ These findings suggest that beta-adrenergic blockade reduces myocardial injury during CPR without decreasing the likelihood of successful defibrillation or compromising spontaneous postresuscitation left ventricular function.