Paediatric anaesthesia
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Paediatric anaesthesia · Jan 2004
ReviewChronic upper airway obstruction and cardiac dysfunction: anatomy, pathophysiology and anesthetic implications.
The causes of obstruction to airflow in the pediatric upper airway include craniofacial disorders, subglottic stenosis, choanal atresia, syndromes associated with neuromuscular weakness, and the most common, hypertrophy of the tonsils and adenoids. Abnormal breathing can adversely affect craniofacial growth, and abnormal craniofacial development can promote upper airway obstruction. Chronic upper airway obstruction often presents with evidence of obstructive sleep apnea syndrome; in severe cases these children also present with pulmonary hypertension and cor pulmonale. ⋯ The anesthetic considerations for children undergoing adenotonsillectomy for chronic airway obstruction are significant. These children are at high risk for complications such as laryngospasm, desaturation, stimulation of pulmonary hypertension and cardiac dysfunction, pulmonary edema, postoperative upper airway obstruction, and respiratory arrest. Because of underlying condition(s) (facial abnormalities, neuromuscular disease, etc.), successful adenotonsillar surgery may not improve upper airway obstruction significantly, especially in the immediate postoperative period when edema, bleeding and the effects of anesthetics and analgesics are present.
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Paediatric anaesthesia · Jan 2004
ReviewTrauma of the larynx and craniofacial structures: airway implications.
Laryngeal trauma in children is potentially life-threatening. An organized approach by all who care for these children in an emergency situation is essential. Prompt recognition and treatment of such injuries will minimize the risk of long-term complications that would require multiple operative procedures and prolonged rehabilitation.
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Paediatric anaesthesia · Jan 2004
ReviewAirway structure, function and development in health and disease.
Until they are fully mature, the airways are highly susceptible to damage. Factors that may contribute to vulnerability of immature airways and the occurrence of bronchopulmonary dysplasia (BPD) in preterm neonates include decreased contractility of smooth muscles of the airway, which leads to generation of lower forces, and immaturity of airway cartilage, leading to increased compressibility of developing airways. ⋯ Imaging has demonstrated an excessively decreased airway cross-sectional area during exhalation in infants with BPD and acquired tracheomegaly in very preterm infants who had received mechanical ventilatory support. To further advance our understanding of how the airways develop, and to design less damaging protocols for mechanical ventilation in preterm neonates, basic laboratory studies of airway ultrastructure need to be performed and the results correlated with clinical pulmonary function studies.
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Paediatric anaesthesia · Nov 2003
Letter Case ReportsAnaesthetic management of an infant with Conradi's syndrome.