Kardiologiya
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Mechanisms involved in the development of parasystole and extrasystole are substantiated on the basis of vast clinical material (261 patients with parasystole and 45 with extrasystole). The use of functional tests employing exercise and atropine contributed to both the correct diagnosis of parasystole and extrasystole and a logical explanation of the mechanisms governing their development. ⋯ The localization of ectopic foci in parasystole and extrasystole confirms the hypothesis of the mechanisms involved in these arrhythmias. A parallel study of parasystole and extrasystole defined parasystole as active heterotopia, and extrasystole, as passive heterotopia incapable of generating a rhythm.
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Review Comparative Study
[Basic principles of the current treatment of primary pulmonary hypertension].
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The authors analyzed the cause of diagnostic errors due to incorrect ECG interpretation. It was revealed that the cause for diagnostic errors may lie in the inadequate experience of the electrocardiologist , the overestimation of the method potentialities and the absence of the commonly accepted language of ECG interpretation. ⋯ The clinical-morphological approach in clinical practice is acceptable only provided that the electrocardiologist is acquainted with the clinical picture of the disease. The use of this approach disregarding the clinical findings constitutes one of the causes of diagnostic errors and iatrogenia .
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The differentiation between linked parasystole and extrasystole was based on functional tests with exercise and atropin as stimuli. A total of 116 patients (71 with parasystole and 45 with extrasystole) were investigated. ⋯ Prolonged ECG recording established limits for the maximum interval of links between monotopic extrasystoles and the interectopic interval for paired extrasystole. Extrasystole was shown to be incapable of generating a pace of its own, i.e. a succession of three or more ectopic complexes of similar morphology.