Articles: subarachnoid-hemorrhage.
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Journal of neurosurgery · Sep 1979
Ventricular dilatation and communicating hydrocephalus following spontaneous subarachnoid hemorrhage.
Ventricular dilatation following spontaneous subarachnoid hemorrhage (SAH) is a well recognized phenomenon. Its clinical significance, however, remains controversial. Two phases are distinguished, the acute or early, occurring soon after the ictus, and the chronic or late, developing after the second week. ⋯ Their findings suggest that ventricular dilatation soon after SAH is not always clinically significant and does not necessarily require shunting before definitive surgery. Delayed symptomatic ventricular enlargement (communicating hydrocephalus) occurs in 7% of the patients and can be safely diagnosed on the basis of the clinical picture and CT scan appearances. Treatment with a ventricular shunting system is almost invariably rewarding.
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The CT findings in patients with subarachnoid hemorrhage include visualization of the aneurysm (10% of the cases), subarachnoid, intraventricular, or intracerebral hemorrhage (87% of cases scanned within five days), "ischemic" lucency secondary to spasm (10%), and hydrocephalus (54%).
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Obstetrics and gynecology · Jan 1979
Case ReportsSubarachnoid hemorrhage secondary to ruptured cerebral aneurysm in pregnancy.
Eight patients suffering ruptured cerebral aneurysms during pregnancy were managed at the University of Western Ontario hospitals between 1967 and 1977. Seven aneurysms were managed surgically. All of these patients survived, 1 with permanent neurologic deficit. ⋯ The prognosis for ruptured cerebral aneurysms during pregnancy is good for both mother and fetus. A short course of conservative therapy followed by surgical management of the aneurysm is advocated. Delivery may be managed according to obstetrical indications following surgical correction of the aneurysm.
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A case of acute pulmonary edema after subarachnoid hemorrhage is presented. A supra and infratentorial arteriovenous malformation was revealed by serial cerebral angiography. Sequential chest films documented regression of both, pulmonary edema and cardiac enlargment. The literature is reviewed and a possible physiopathology is discussed.