• Critical care clinics · Jan 1991

    Review Case Reports

    Endocrine crises. Hypophosphatemia and hyperphosphatemia.

    • M P Peppers, M Geheb, and T Desai.
    • Department of Pharmacy, Mineral Area Regional Medical Center, Farmington, Missouri.
    • Crit Care Clin. 1991 Jan 1; 7 (1): 201-14.

    AbstractPathophysiology, clinical sequelae, and treatment for hypophosphatemia and hyperphosphatemia are discussed. Hypophosphatemia results from a variety of conditions including malnutrition, carbohydrate refeeding, acid-base disorders, and hormonal and drug effects. Patients suffering from severe hypophosphatemia may present with a variety of syndromes that can become detrimental and even life-threatening if this electrolyte depletion goes untreated. Hyperphosphatemia occurs because of increased extracellular phosphate load from either endogenous or exogenous sources or from a decrease in renal phosphate excretion. Left untreated, hyperphosphatemia can result in dangerous calciumphosphate precipitation into vital organs and tissues.

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