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Neurobiology of aging · Aug 2011
The wake-promoting effects of hypocretin-1 are attenuated in old rats.
- Stephen R Morairty, Jonathan Wisor, Kristy Silveira, William Sinko, and Thomas S Kilduff.
- Biosciences Division, SRI International, CA 94025, USA. stephen.morairty@sri.com
- Neurobiol. Aging. 2011 Aug 1; 32 (8): 1514-27.
AbstractDisruption of sleep is a frequent complaint among elderly humans and is also evident in aged laboratory rodents. The neurobiological bases of age-related sleep/wake disruption are unknown. Given the critical role of the hypocretins in sleep/wake regulation, we sought to determine whether the wake-promoting effect of hypocretin changes with age in Wistar rats, a strain in which age-related changes in both sleep and hypocretin signaling have been reported. Intracerebroventricular infusions of hypocretin-1 (10 and 30 μg) significantly increased wake time relative to vehicle in both young (3 mos) and old (25 mos) Wistar rats. However, the magnitude and duration of the wake-promoting effects were attenuated with age. An increase of parameters associated with homeostatic sleep recovery after sleep deprivation, including non-rapid eye movement (NR) sleep time, NR delta power, the ratio of NR to rapid eye movement (REM) sleep, and NR consolidation, occurred subsequent to Hcrt-induced waking in young but not old rats. ICV infusions of hypocretin-2 (10 and 30 μg) produced fewer effects in both young and old rats. These data demonstrate that activation of a major sleep/wake regulatory pathway is attenuated in old rats.Copyright © 2009 Elsevier Inc. All rights reserved.
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