• Neurology · Sep 2005

    Lack of beta-methylamino-l-alanine in brain from controls, AD, or Chamorros with PDC.

    • Thomas J Montine, Ke Li, Daniel P Perl, and Douglas Galasko.
    • Department of Pathology, University of Washington, Seattle, WA 98104, USA. tmontine@u.washington.edu
    • Neurology. 2005 Sep 13; 65 (5): 768-9.

    AbstractHigh levels of beta-methylamino-l-alanine (BMAA), a putative neurotoxin, have been reported in brain samples from Chamorros and patients with Alzheimer disease (AD) from western Canada. The authors assayed free BMAA in the brains of five control subjects and five patients with AD from the US Pacific Northwest as well as Chamorros with and without Parkinson-dementia complex. In contrast to others, they detected no free BMAA in any of these samples.

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