• Resp Res · Jan 2013

    Comment Letter

    Role of epithelial mesenchymal transition (EMT) in chronic obstructive pulmonary disease (COPD).

    • Sukhwinder Singh Sohal and Eugene Haydn Walters.
    • NHMRC Centre of Research Excellence for Chronic Respiratory Disease, School of Medicine, University of Tasmania, Hobart 7000, Australia. haydn.walters@utas.edu.au.
    • Resp Res. 2013 Jan 1;14:120.

    AbstractSmall airway fibrosis is the main contributor to physiological airway dysfunction in COPD. One potential mechanism contributing to small airway fibrosis is epithelial mesenchymal transition (EMT). When associated with angiogenesis (so called EMT-Type-3) it may well also be the link with the development of cancer, which is closely associated with COPD and predominantly in large airways. In a recent study published in Respiratory Research, Qin Wang and colleagues investigated the role of urokinase plasminogen activator receptor (uPAR) in EMT in small airway epithelium of COPD patients. However, there are some issues with the paper which we wish to comment on.

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