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- Natochin YuV Sechenov Institute of Evolutionary Physiology & Biochemistry, Sankt-Petersburg, Russian Federation. and E I Shakhmatova.
- Sechenov Institute of Evolutionary Physiology & Biochemistry, Sankt-Petersburg, Russian Federation.
- Pflugers Arch. 1992 Jul 1;421(4):406-8.
AbstractV1-antagonist, [Mca1, D-Phe2, Sar7] arginine vasopressin, at a concentration 0.5 nM to 5 nM increased hydroosmotic effect of 5 nM arginine vasopressin in frog urinary bladder. 1 nM V1-antagonist [Mca1, O-Me-Tyr2] arginine vasopressin does not changed hydroosmotic effect of 0.1 nM dibutyryl cAMP. We suggest that-both V1- and V2-receptors are present on epithelial cells of frog urinary bladder and that V1-receptors are involved in the modulation of the hydroosmotic effect of arginine vasopressin-mediated by V2-receptors. by V2-receptors.
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