• Am. J. Cardiol. · Dec 2005

    Comparative Study

    Effect of vasopressin on hemodynamics in patients with refractory cardiogenic shock complicating acute myocardial infarction.

    • Sanjit Jolly, Gary Newton, Eric Horlick, Peter H Seidelin, Heather J Ross, Mansoor Husain, and Vladimir Dzavik.
    • The Interventional Cardiology Program, Division of Cardiology, University Health Network, University of Toronto, Toronto, Ontario, Canada.
    • Am. J. Cardiol. 2005 Dec 15;96(12):1617-20.

    AbstractIn a retrospective study of 36 patients who developed cardiogenic shock after myocardial infarction, intravenous vasopressin therapy increased mean arterial pressure from 56 to 73 mm Hg at 1 hour (p < 0.001) and maintained it for 24 hours without changing pulmonary capillary wedge pressure, cardiac index, urine output, or other inotropic requirements. After norepinephrine administration, mean pulmonary capillary wedge pressure increased at 1 hour from 21 to 24 mm Hg (p = 0.04); however, this increase was not sustained at 12 and 24 hours. Norepinephrine was associated with a significant increase in cardiac power index at 24 hours, whereas there was only a trend for an increase in cardiac power with vasopressin therapy. In a cohort of patients who developed refractory cardiogenic shock after myocardial infarction, vasopressin was associated with increased mean arterial pressure and no adverse effect on other hemodynamic parameters.

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