• Aline Mizuta Kozoroski Kanashiro, Paula Levatti Alexandre, Cristiana Borges Pereira, Antonio Carlos de Paiva Melo, and Milberto Scaff.
• Serviço de Neurologia, Hospital do Servidor Público Estadual de São Paulo, São Paulo, SP, Brazil. kozoroski@terra.com.br
• Arq Neuropsiquiatr. 2005 Sep 1;63(3A):643-7.

AbstractVestibular paroxysmia is a syndrome of cross-compression of the VIII cranial nerve and was first described by Jannetta who used the term "disabling positional vertigo". This syndrome is characterized by brief attacks of vertigo, tinnitus, vestibular and auditory deficits. MRI may show the VIII nerve compression from vessels in the posterior fossa, such as the basilar, vertebral, anterior-inferior cerebellar or the posterior-inferior cerebellar arteries. Vestibular paroxysmia may be treated either with medical therapy, such as carbamazepine, phenytoin or gabapentin or with the microvascular decompression of the VIII nerve. This study describes eight patients with vestibular paroxysmia. Four of them showed also clinical signs suggesting cross-compression of the V and/or VII nerve. Seven patients treated with carbamazepine had significant improvement of vertigo and tinnitus.

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