• Richard Nolz, Harald Teufelsbauer, Ulrika Asenbaum, Dietrich Beitzke, Martin Funovics, Andreas Wibmer, Christina Plank, Alexander M Prusa, Johannes Lammer, and Maria Schoder.
• Department of Radiology, Division of Cardiovascular and Interventional Radiology, Medical University of Vienna, Austria. richard.nolz@meduniwien.ac.at
• J. Endovasc. Ther. 2012 Apr 1;19(2):193-9.

PurposeTo evaluate the frequency of type II endoleaks after endovascular aneurysm repair (EVAR) and to compare sac diameter and neck changes in patients with type II endoleak to endoleak-free patients with at least 3-year imaging follow-up.MethodsAmong 407 consecutive EVAR patients, 109 patients (101 men; mean age 72.1 years, range 55-86) had at least 3-year computed tomography (CT) data and no type I or III endoleak. In this cohort, 49 patients presented with a type II endoleak at some time and 60 patients had no endoleak. Patients with type II endoleaks were further divided into subgroups based on the vessel origin and the perfusion status (persistent or transient). The course of the perfusion status of type II endoleaks and changes in the aneurysm sac diameters, neck diameters, and renal to stent-graft distances (RSD) were evaluated in the defined groups. Reintervention and death rates were also reported.ResultsThe mean follow-up was 68.1 ± 23.8 months. Compared to the no endoleak group, overall sac diameter increased significantly in the type II endoleak group (p = 0.007), but vessel origin did not have any influence. With regard to the perfusion status of type II endoleaks, aneurysm sac changes were significantly higher (p = 0.002) in the persistent endoleak group. During the study period, the increase in the proximal neck diameter was significantly higher in the no endoleak group compared to the type II endoleak group (p = 0.025). No significant difference was found in RSD changes between the defined groups. Reinterventions were performed in 20 (18.3%) patients (13 for type II endoleak); 2 (1.8%) patients without type II endoleak died of ruptured aneurysm.ConclusionPersistent type II endoleaks led to significant aneurysm sac enlargement, but without increased mortality or rupture rates.

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