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- K A Sowards.
- Department of Sociology, Washington State University, Pullman 99164-4020, USA. sowardsk@vancouver.wsu.edu
- Demography. 1997 Aug 1;34(3):399-409.
AbstractLinked death and birth records from San Antonio, Texas revealed that infectious infant mortality is increasingly a function of premature birth and low birth weight. Between 1935 and 1944, 4% of infectious infant deaths had associated causes involving prematurity and related conditions; by 1980, 25% of infectious infant deaths involved prematurity and more than 40% of those infants weighed less than 2,500 grams. The shift in birth-weight composition results almost entirely from an increase in very low-weight births. Under conditions of advanced perinatal technology, infectious infant mortality should no longer be viewed as wholly exogenous. These findings further undermine the contemporary relevance of the exogenous-endogenous distinction.
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