• Corey R Fehnel, Linda C Wendell, N Stevenson Potter, Petra Klinge, and Bradford B Thompson.
• Rhode Island Hospital Division of Neurocritical Care, Assistant Professor of Neurology and Neurosurgery, The Alpert Medical School of Brown University.
• R I Med J (2013). 2014 Jul 1; 97 (7): 45-6.

AbstractSevere traumatic brain injury is associated with both acute and delayed neuro- logical injury. Cerebral vasospasm is commonly associated with delayed neurological decline in aneurysmal subarachnoid hemorrhage patients. However, the role played by vasospasm in traumatic brain injury is less clear. Vasospasm occurs earlier, for a shorter duration, and often without significant neurological consequence among traumatic brain injury patients. Detection and management strategies for vasospasm in aneurysmal subarachnoid hemorrhage are not easily transferrable to traumatic brain injury patients. We present a patient with a severe traumatic brain injury who had dramatic improvement following emergent decompressive hemicraniectomy. Two weeks after initial presentation he suffered a precipitous decline despite intensive surveillance. This case illustrates the distinct challenges of diagnosing cerebral vasospasm in the setting of severe traumatic brain injury.

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