• Shock · Jun 2017

    Endothelial Damage Signals Refractory Acute Kidney Injury in Critically Ill Patients.

    • Theis S Itenov, Jens-Ulrik Jensen, Sisse R Ostrowski, Pär I Johansson, Katrin M Thormar, Jens D Lundgren, Morten H Bestle, and “Procalcitonin And Survival Study” study group.
    • *Department of Anesthesiology, Nordsjællands Hospital, University of Copenhagen, Copenhagen, Denmark†CHIP/Department of Infectious Diseases at Rigshospitalet and the University of Copenhagen, Copenhagen, Denmark‡Department of Clinical Microbiology, Hvidovre Hospital, University of Copenhagen, Copenhagen, Denmark§Section for Transfusion Medicine, Capital Region Blood Bank, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark||Division of Acute Care Surgery, Department of Surgery, Centre of Translational Injury Research (CeTIR), University of Texas Health Medical School at Houston, Houston, Texas¶Department of Anesthesiology, Bispebjerg Hospital, Copenhagen, Denmark.
    • Shock. 2017 Jun 1; 47 (6): 696-701.

    AbstractCritically ill patients with acute kidney injury (AKI) are heterogeneous on pathophysiology and prognosis. The role of endothelial damage in the pathogenesis of refractory AKI has not been clarified. The aim was to determine if biomarkers of endothelial damage, independently of the inflammatory insult on the kidney, can predict recovery of acute kidney injury.

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