• World Neurosurg · Jan 2019

    Central or axial atlantoaxial dislocation as a cause of cervical myelopathy: a report of outcome of 5 cases treated by atlantoaxial stabilization.

    • Atul Goel, Arjun Dhar, Abhidha Shah, Dikpal Jadhav, Nilesh Bakale, Tejas Vaja, and Neha Jadhav.
    • Department of Neurosurgery, K.E.M. Hospital and Seth G.S. Medical College, Parel, Mumbai, India; Department of Neurosurgery, Lilavati Hospital and Research Centre, Bandra (E), Mumbai, India. Electronic address: atulgoel62@hotmail.com.
    • World Neurosurg. 2019 Jan 1; 121: e908-e916.

    ObjectiveThe implications of diagnosis and treatment of central or axial atlantoaxial dislocation (CAAD) as a cause of symptoms of cervical myelopathy are evaluated.MethodsThis is a report of a series of 5 patients who presented with the primary symptoms of spasticity and motor weakness and paresthesias in all the limbs. There was no evidence of any significant compression of the dural tube or neural structures at the craniovertebral junction. There was no craniovertebral junction instability when assessed by classically described radiologic parameters. CAAD was diagnosed based on our recently discussed parameter of facetal alignment, corroborative clinical and radiologic evidence, and direct observation of atlantoaxial instability by manual manipulation of the bones of the region. All patients underwent atlantoaxial fixation.ResultsThere was remarkable improvement in the clinical symptoms in the immediate postoperative period and during the follow-up period of 12-24 months (average, 16 months). All patients have continued to have progressive clinical recovery. Clinical assessments were done using Goel clinical evaluation scale, Japanese Orthopedic Association score, and visual analog scale.ConclusionsIdentification and treatment of CAAD can have a significant management impact on patients where the cause of spastic quadriparesis is otherwise undiagnosed.Copyright © 2018 Elsevier Inc. All rights reserved.

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