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- A Hara, M Niwa, T Iwai, H Yano, M Nakashima, Y Bunai, T Uematsu, N Yoshimi, and H Mori.
- Department of Pathology, Gifu University School of Medicine, Gifu, Japan. ahara@cc.gifu-u.ac.jp
- Brain Res. 1999 Sep 4; 840 (1-2): 167-70.
AbstractPost-ischemic administration of 2-deoxy-D-glucose (2-DG), a glucose antimetabolite, markedly reduces the occurrence of ischemia-induced delayed neuronal death (DND) in the gerbil hippocampus. This means that the reduction of energy dependent metabolism after ischemia prevents ischemia-induced damages of hippocampal neurons. In the present study, we demonstrated hyperthermia during ischemia fails to preserve neurons in hippocampal CA1 of 2-DG treated gerbil following transient forebrain ischemia.
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