Endovascular treatment of cerebral vasospasm: vasodilators

Neuroimaging Clin. N. Am. · Nov 2013

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Endovascular treatment of cerebral vasospasm: vasodilators and angioplasty.

Cerebral vasospasm following aneurysmal subarachnoid hemorrhage (SAH) is a delayed, reversible narrowing of the intracranial vasculature that occurs most commonly 4 to 14 days after aneurysmal SAH and can lead to permanent ischemic injury. Angiographic spasm occurs in up to 70% of patients following SAH, and approximately half become symptomatic. Estimates of patients who are disabled by vasospasm, or die because of it, range from 5% to 9%, with vasospasm accounting for 12% to 17% of all fatalities or cases of disability after SAH. This article discusses the multiple medical and endovascular therapies used to prevent or treat vasospasm.

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- Aditya S Pandey, Augusto E Elias, Neeraj Chaudhary, Byron G Thompson, and Joseph J Gemmete.
- Division of Interventional Neuroradiology, Departments of Neurosurgery and Radiology, University of Michigan Hospitals, 1500 East Medical Center Drive, Ann Arbor, MI 48109-5030, USA.
- Neuroimaging Clin. N. Am.. 2013 Nov 1;23(4):593-604.
AbstractCerebral vasospasm following aneurysmal subarachnoid hemorrhage (SAH) is a delayed, reversible narrowing of the intracranial vasculature that occurs most commonly 4 to 14 days after aneurysmal SAH and can lead to permanent ischemic injury. Angiographic spasm occurs in up to 70% of patients following SAH, and approximately half become symptomatic. Estimates of patients who are disabled by vasospasm, or die because of it, range from 5% to 9%, with vasospasm accounting for 12% to 17% of all fatalities or cases of disability after SAH. This article discusses the multiple medical and endovascular therapies used to prevent or treat vasospasm.Copyright © 2013 Elsevier Inc. All rights reserved.

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Endovascular treatment of cerebral vasospasm: vasodilators and angioplasty.

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