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Critical care clinics · Apr 2020
ReviewImmune Consequences of Endothelial Cells' Activation and Dysfunction During Sepsis.
- Stéphanie Pons, Marine Arnaud, Maud Loiselle, Eden Arrii, Elie Azoulay, and Lara Zafrani.
- INSERM U976, Saint-Louis Teaching Hospital, 1, Avenue Claude Vellefaux, Paris 75010, France.
- Crit Care Clin. 2020 Apr 1; 36 (2): 401-413.
AbstractThe vascular endothelium provides a direct interface between circulating blood cells and parenchymal cells. Thus, it has a key role in vasomotor tone regulation, primary hemostasis, vascular barrier, and immunity. In the case of systemic inflammation, endothelial cell (EC) activation initiates a powerful innate immune response to eliminate the pathogen. In some specific conditions, ECs may also contribute to the activation of adaptive immunity and the recruitment of antigen-specific lymphocytes. However, the loss of EC functions or an exaggerated activation of ECs during sepsis can lead to multiorgan failure.Copyright © 2019 Elsevier Inc. All rights reserved.
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