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- Ajit Lalvani, Marcel A Behr, and Saranya Sridhar.
- Tuberculosis Research Unit, National Heart and Lung Institute, Imperial College London, Norfolk Place, London W2 1PG, UK. a.lalvani@imperial.ac.uk
- Cell. 2012 Feb 3; 148 (3): 389-91.
AbstractTobin and colleagues show that both inhibition and excessive production of the inflammatory mediator TNFα impact the pathogenesis of tuberculosis (TB) and the response to therapy. Identifying a critical role for the genetically determined balance between pro- and anti-inflammatory eicosanoids in regulating TNFα levels provides a roadmap to tailored TB treatment based on host genotype.Copyright © 2012 Elsevier Inc. All rights reserved.
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