Journal of neurology, neurosurgery, and psychiatry
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J. Neurol. Neurosurg. Psychiatr. · Apr 2004
Email triage of new neurological outpatient referrals from general practice.
To determine whether an email triage system between general practitioners and a neurologist for new outpatient referrals is feasible, acceptable, efficient, safe, and effective. ⋯ Email triage is feasible, acceptable to GPs, and safe. It has the potential for making the practice of neurologists more efficient, and this needs to be tested in a larger randomised study.
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J. Neurol. Neurosurg. Psychiatr. · Apr 2004
Religiosity is associated with hippocampal but not amygdala volumes in patients with refractory epilepsy.
To assess the relationship between the behavioural triad of hyper-religiosity, hypergraphia and hyposexuality in epilepsy, and volumes of the mesial temporal structures. ⋯ These findings suggest that right hippocampal volumes are negatively correlated with religiosity in patients with refractory epilepsy.
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J. Neurol. Neurosurg. Psychiatr. · Apr 2004
Inhibition of ongoing responses in patients with Parkinson's disease.
We investigated the involvement of the basal ganglia in inhibiting ongoing responses in patients with Parkinson's disease (PD). ⋯ Our results provide evidence for involvement of the basal ganglia in the inhibition of ongoing responses.
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J. Neurol. Neurosurg. Psychiatr. · Apr 2004
Motor cortex hyperexcitability to transcranial magnetic stimulation in Alzheimer's disease.
Recent transcranial magnetic stimulation (TMS) studies demonstrate that motor cortex excitability is increased in Alzheimer's disease (AD) and that intracortical inhibitory phenomena are impaired. The aim of the present study was to determine whether hyperexcitability is due to the impairment of intracortical inhibitory circuits or to an independent abnormality of excitatory circuits. ⋯ The change in threshold did not seem to correlate with dysfunction of inhibitory intracortical cholinergic and GABAergic circuits, nor with the central cholinergic activity. We propose that the hyperexcitability of the motor cortex is caused by an abnormality of intracortical excitatory circuits.