Journal of vascular surgery
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Review Case Reports
Saccular aneurysm in the right-sided aortic arch: a successfully corrected case.
We report the surgical treatment of a rare case of true aortic aneurysm in a right-sided aortic arch. A 49-year-old female patient with obstructive respiratory problems demonstrated a true aneurysm with a diameter of 58 mm located at the right-sided aortic arch between the right carotid artery and right subclavian artery. ⋯ The positions of true aneurysms in the right-sided aortic arch can be divided into two subtypes: first, the transverse arch between the right carotid artery and right subclavian artery, and second, the base of the subclavian artery, the Kommerell's diverticulum. The region is informative for consideration of the surgical approach toward aneurysms of this entity.
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Comparative Study
Aminoterminal propeptide of type III procollagen in the follow-up of patients with abdominal aortic aneurysms.
We evaluate here whether serial changes in the concentration of the aminoterminal propeptide of type III procollagen (PIIINP) in serum bear any relationship to the rate of abdominal aortic aneurysm (AAA) expansion and whether serum PIIINP has any predictive value with respect to the rupture event. ⋯ Acceleration of AAA growth is reflected in serum PIIINP, and a marked elevation of serum PIIINP during follow-up of a patient with an AAA may predict an approaching rupture event.
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Comparative Study
Continuous electroencephalographic monitoring and selective shunting reduces neurologic morbidity rates in carotid endarterectomy.
The role of continuous electroencephalographic (EEG) monitoring during carotid endarterectomy was evaluated in this retrospective review. ⋯ The overall neurologic morbidity rate was significantly lower in the EEG group than in the non-EEG group, therapy demonstrating the value of intraoperative EEG monitoring in carotid endarterectomy.
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The goal of this study was to determine the effects of vasopressin and the selective V2-receptor agonist desmopressin on human saphenous veins, with special emphasis on endothelium-mediated responses. ⋯ These observations indicate that vasopressin exerts contractile effects on human saphenous vein by V1-receptor stimulation. Vasopressin causes dilatation of human saphenous vein only if V1-receptor blockade is present. This relaxation appears to be mediated by the release of relaxant prostaglandins, probably derived from endothelial cells, and is independent of V2-receptor stimulation or release of nitric oxide. Desmopressin elicits relaxation that is largely dependent on V2-receptor stimulation, which may bring about the release of dilating prostaglandins from the endothelial cells.