Journal of internal medicine
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The above letter to the editor written by "Lars Andersson" (stated to be affiliated with Department of Physiology and Pharmacology, Karolinska Institutet), published in print and online in the Journal of Internal Medicine in Wiley Online Library https://onlinelibrary.wiley.com) on the date given above, has been retracted by agreement between the journal's Editor-in-Chief Ulf de Faire, the Association for Publication of the Journal of Internal Medicine and John Wiley & Sons Limited. The retraction has been agreed due to the author's use of what appears to be a false name and affiliation. ⋯ Response on the author's reply to the Letter to the Editor: contradictory data on type 1 diabetes in a recently published article: risks of neurological and immune-related diseases, including narcolepsy, after vaccination with Pandemrix. J Intern Med 2015; 277: 272-3.
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Whether a causal relationship exists between milk intake and reduced risk of fractures is unclear. ⋯ Genetically lifelong lactase persistence with high milk intake was not associated with hip fracture in Danish population-based cohorts. A meta-analysis combining Danish studies with published Northern European population studies also showed that lactase persistence was not associated with fracture risk. Genetic lactase persistence was associated with a higher femoral neck BMD, but not lumbar spine or total hip BMD.
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Multicenter Study Observational Study
Hyponatremia and activation of vasopressin secretion are both independently associated with 30-day mortality: results of a multicenter, observational study.
Hyponatremia is a common feature of acute illness and associated with increased mortality. This may be explained by a stress-mediated activation of the vasopressin system with an increase in free-water reabsorption. ⋯ This prospective study including medical patients upon emergency room admission found hyponatremia as well as an activation of the vasopressin system to be independently associated with mortality. This suggests that stress- and vasopressin-independent mechanisms are responsible for the association of low sodium levels with mortality.