Journal of anesthesia
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Journal of anesthesia · Jun 1998
Continuous measurement of hematocrit using an intravascular catheter equipped with a fiberoptic transmission cell.
The purpose of this study was to measure intravascular hematocrit values continuously by using a fiberoptic probe based on near-infrared photometry. ⋯ This method is effective for monitoring the rapid changes in hematocrit.
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Journal of anesthesia · Jun 1998
Effect of anesthetics on the self-sustained oscillation in an artificial membrane induced by repetitive conformational change of DOPH molecules between hydrophilic and hydrophobic phases.
The mechanism of anesthesia was approached from a study of an artificial excitable membrane that well reproduced the active electrical properties of the nerve membrane. ⋯ The volatile anesthetic agents affected the hydrophobic oil droplets of the DOPH molecules and impeded their repetitive conformational change between the hydrophilic and hydrophobic phases, just as local anesthetics had been reported to do.
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Journal of anesthesia · Jun 1998
Comparative evaluation of TIVA with propofol-fentanyl and thiopental-sevoflurane anesthesia using laryngeal mask airway for diagnostic bronchoscopy.
Diagnostic bronchoscopy is performed under general anesthesia in our hospital. This study was designed to determine whether total intravenous anesthesia (TIVA) with propofol-fentanyl provides more stable hemodynamics using a laryngeal mask airway (LMA) for diagnostic bronchoscopy than thiopental-sevoflurane anesthesia. ⋯ TIVA with propofol-fentanyl in conjunction with an LMA performs better than thiopental-sevoflurane anesthesia for diagnostic bronchoscopy because of its superior maintenance of cardiovascular stability.
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Journal of anesthesia · Jun 1998
Attenuation of nitric oxide-stimulated soluble guanylyl cyclase from the rat brain by halogenated volatile anesthetics.
The present study was undertaken to examine whether interaction between halogenated volatile anesthetics and nitric oxide (NO) at soluble guanylyl cyclase (sGC) would occur in rat brain. ⋯ These results suggest that halogenated volatile anesthetics can attenuate the activity of NO-stimulated sGC by competing with NO for the NO binding site on the enzyme.