Journal of neurosurgical anesthesiology
-
J Neurosurg Anesthesiol · Jul 2010
Accreditation and standardization of neuroanesthesia fellowship programs: results of a specialty-wide survey.
The question of accreditation and standardization of neuroanesthesia fellowship training programs in the U. S. has been discussed extensively within the field. Although numerous opinion pieces have been published, there are no data indicating the level of support or opposition for accreditation of subspecialty training among specialists in the field of neuroanesthesia. ⋯ Career development, neurocritical care, and intraoperative neuromonitoring were the top 3 subjects thought to be essential to a neuroanesthesia fellowship. The majority supported a 1-year fellowship training program. These data indicate measurable support among members of SNACC for a process toward the accreditation of neuroanesthesia fellowship training programs.
-
J Neurosurg Anesthesiol · Jul 2010
The efficacy of motor-evoked potentials on cerebral aneurysm surgery and new-onset postoperative motor deficits.
Surgical clipping may cause stenosis of parent arteries or occlusion of perforating arteries in cerebral aneurysm surgery. To prevent postoperative motor deficits, motor-evoked potentials (MEPs) have been used. This enables to detect cerebral ischemia. ⋯ Only 1 of the 6 patients had aneurysm in AchA. In AchA aneurysm surgery, intraoperative MEP monitoring seems to be useful. False negative in MEP monitoring may include new-onset hemiparesis despite preserved intraoperative MEP.
-
J Neurosurg Anesthesiol · Jul 2010
The effect of sevoflurane on neuronal degeneration and GABAA subunit composition in a developing rat model of organotypic hippocampal slice cultures.
The GABA(A) receptor subunit composition undergoes a switch from a predominantly alpha2 to a predominantly alpha1 around postnatal day (PND) 7 in a rat pup. This developmental switch in the GABA(A) receptor subunit composition changes the kinetics and pharmacologic properties of the GABA(A) receptor. Using a developmental organotypic hippocampal slice model, we hypothesized that the developmental changes in the GABA(A) receptor subunit composition may promote neurodegeneration after exposure to sevoflurane. ⋯ This in vitro investigation supports an age-dependent and GABA(A) receptor subunit composition relationship between 2.0% sevoflurane exposure and cell death.