Der Anaesthesist
-
Environmental contamination with bacteria in the course of extubation was shown to be minimal as compared to contamination of the hands and gowns. 7 out of 21 anaesthesiologists were carriers of staphylococcus aureus. Two persons spread staphylococci to their patients and the operating room environment in spite of strict adherence to conventional hygienic policies.
-
Case Reports
[Permeability pulmonary edema (neurogenic pulmonary edema) following isolated head and brain injury].
This paper reports on 3 patients with permeability pulmonary oedema accompanying an isolated head injury (neurogenic pulmonary oedema - NPE). The occurrence of a NPE in our patients with isolated head injury amounts to 0.62%. Comparing our case reports and results, respectively, with those published in the literature, the pathogenesis of NPE is discussed and delineated.
-
There is an increase in endotracheal tube-cuff volume and pressure due to nitrous oxide diffusion into the cuff during anaesthesia. A rise of the cuff pressure up to 100 mm Hg (high volume-low pressure cuffs!) within only two hours is nothing out of the ordinary. The inspiratory nitrous oxide concentration influences the cuff pressure rises. ⋯ In endotracheal tubes with a Rediffusion System, cuff pressure never exceeds capillary perfusion pressure of the tracheal mucosa. In our in vitro-experiments we found an increase of cuff pressure from 14.2 +/- 0.5 mm Hg to only 27.3 +/- 1.9 mm Hg within six hours (FIN2O = 0.66). Within 150 minutes of endotracheal anaesthesia (FIN2O = 0.66) cuff pressure rose from 14.6 +/- 0.5 mm Hg to only 21.5 +/- 3.6 mm Hg.(ABSTRACT TRUNCATED AT 250 WORDS)