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- Sarah Inés Ramírez, Elizabeth Ashley Suniega, and Megan Ilene Laughrey.
- Department of Family and Community Medicine, Penn State College of Medicine, 500 University Drive, HP11, Hershey, PA 17033, USA. Electronic address: sramirez2@pennstatehealth.psu.edu.
- Prim. Care. 2024 Sep 1; 51 (3): 535547535-547.
AbstractDisequilibrium of hormonal intercommunication between the maternal brain and the developing fetal-placental unit increases morbidity and mortality risk for the mother-baby dyad. As a novel yet temporary endocrine organ, the placenta serves as a physical and immunologic barrier that facilitates exchange of nutrients and elimination of fetal waste. Steroid and peptide-based hormones secreted by the placenta and other neuroendocrine organs induce adaptations in maternal physiology accommodating fetal growth and development and enabling lactation postpartum. Human placental growth hormone, a peptide hormone continuously secreted at increasing concentrations throughout pregnancy, is a primary determinant of maternal insulin resistance and gestational diabetes.Copyright © 2024 Elsevier Inc. All rights reserved.
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